I feel at a loss as to help renaeden in this issue as I know little about stimming (except where it concerns myself, in a typical autistic fashion), or tics. So I did a little bit of research and came up with the following. (I found out I have
Palilalia though)
(boldedness added by me to signify significance..yeah)
Donna Williams wrote:Stimming stood for 'self stimulatory behaviours'. Something non-auties
imagined they didn't have!
Stimming is aword created by non-auties with often negative connotations.
It was a word created by non-auties who didn't understand that some folks
had compulsive but involuntary tics, some had self calming tools they didn't
understand, some had sensory fascinations they couldn't relate to, some
needed to tune out to tune in, some needed a tool for social distance in the
face of compulsive social invaders, some needed to be repetitive in order to
download, some needed to compensate for a non-autie multitrack world they
couldn't process in thier mono-tracked reality, some needed something to get
lost in when utterly blowing all fuses.... and, anyway, the non-auties
labelled, as is there tendency, this vast array of experiences with one word
as if it was one thing at all times to all people labelled 'Autistic'.
Forgive them. They know not what they see.
Wikipedia wrote:Many theories exist as to what function stimming has, and the reasons for its increased incidence in autistic people. For hyposensitive people, it may provide needed nervous system arousal, releasing beta-endorphins. For hypersensitive people, it may provide a "norming" effect, allowing the person to control a specific part of their sensorium, and is thus a soothing behavior.
Wikipedia also wrote:Sometimes self-injury is viewed as a form of stimming. Usually, self-injury is very different from stimming, but people with decreased pain sensitivity may injure themselves because they like the feel of it, similar to other stims. For example, they might like the way their hand feels in the mouth when they bite themselves, while not feeling the pain of the bite. Or they might like pressure on their forehead and bang their head without it hurting, even if they are risking brain damage.
That's in relation to myself..I found it intriguing.
On the subject of Tics, Wikipedia wrote:Simple motor tics are typically sudden, brief, meaningless movements, such as eye blinking or shoulder shrugging. Motor tics can be of an endless variety and may include such movements as hand-clapping, neck stretching, mouth movements, head, arm or leg jerks, and facial grimacing.
Complex motor tics are typically more purposeful-appearing and of a longer nature. Examples of complex motor tics are pulling at clothes, touching people, touching objects, echopraxia and copropraxia.
Complex tics are rarely seen in the absence of simple tics.
And of the treatment of tics occuring with ADHD, Wikipedia wrote:Treatment of ADHD in the presence of tic disorders
Referred (clinical) populations of patients with Tourette's have a high rate of comorbid ADHD, so the treatment of ADHD co-occurring with tics is often part of the clinical treatment of Tourette's. The treatment of attention-deficit hyperactivity disorder (ADHD) in the presence of tic disorders has long been a controversial topic. Past medical practice held that stimulants could not be used in the presence of tics, due to concern that their use might exacerbate underlying tic disorders. However, multiple lines of research have shown that stimulants can be cautiously used in the presence of tic disorders. Several studies have shown that stimulants do not exacerbate tics any more than placebo does, and suggest that stimulants may even reduce tic severity.
Controversy remains, and the PDR continues to carry a warning that stimulants should not be used in the presence of tic disorders, so physicians may be reluctant to use them. Others are comfortable using them and even advocate for a stimulant trial when ADHD co-occurs with tics, because the symptoms of ADHD can be more impairing than tics.
The stimulants are the first line of treatment for ADHD, with proven efficacy, but they do fail in up to 20% of cases, even in patients without tic disorders. Current prescribed stimulant medications include: methylphenidate (Ritalin®, Metadate®, Concerta®), dextroamphetamine (Dexedrine®), and mixed amphetamine salts (Adderall®). Other medications can be used when stimulants are not an option. These include the alpha-2 agonists (clonidine and guanfacine), tricyclic antidepressants (desipramine and nortriptyline), and newer antidepressants (bupropion, venlafaxine and atomoxetine). There have been case reports of tic exacerbations with bupropion (brand name Wellbutrin®). There is good empirical support for the use of desipramine, bupropion and atomoxetine (brand name Strattera®). Atomoxetine is the only non-controlled Food and Drug Administration (FDA) approved drug for the treatment of ADHD, but is less effective than stimulants for ADHD; is associated with individual cases of liver damage; carries an FDA black box warning regarding suicidal ideation; and controlled studies show increases in heart rate, decreases of body weight, decreased appetite and treatment-emergent nausea.